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丹参酮ⅡA对脑缺血后炎性反应影响机制的研究进展

冯晶晶 李铁军 章越凡

冯晶晶, 李铁军, 章越凡. 丹参酮ⅡA对脑缺血后炎性反应影响机制的研究进展[J]. 药学实践与服务, 2018, 36(2): 108-111. doi: 10.3969/j.issn.1006-0111.2018.02.003
引用本文: 冯晶晶, 李铁军, 章越凡. 丹参酮ⅡA对脑缺血后炎性反应影响机制的研究进展[J]. 药学实践与服务, 2018, 36(2): 108-111. doi: 10.3969/j.issn.1006-0111.2018.02.003
FENG Jingjing, LI Tiejun, ZHANG Yuefan. Progress of tanshinone ⅡA on the mechanism of inflammatory response after cerebral ischemia[J]. Journal of Pharmaceutical Practice and Service, 2018, 36(2): 108-111. doi: 10.3969/j.issn.1006-0111.2018.02.003
Citation: FENG Jingjing, LI Tiejun, ZHANG Yuefan. Progress of tanshinone ⅡA on the mechanism of inflammatory response after cerebral ischemia[J]. Journal of Pharmaceutical Practice and Service, 2018, 36(2): 108-111. doi: 10.3969/j.issn.1006-0111.2018.02.003

丹参酮ⅡA对脑缺血后炎性反应影响机制的研究进展

doi: 10.3969/j.issn.1006-0111.2018.02.003
基金项目: 浦东新区卫生系统重要薄弱学科建设资助(PWZbr2017-16)

Progress of tanshinone ⅡA on the mechanism of inflammatory response after cerebral ischemia

  • 摘要: 丹参酮ⅡA是从丹参中提取的一种菲醌类衍生物,是丹参发挥脑缺血损伤保护药理作用的主要活性成分之一。炎症在脑缺血发病机制中具有重要作用。近年来许多证据表明,免疫细胞、黏附分子、炎性介质等通过诱导或调控脑缺血时中枢神经系统内免疫应答参与发病过程。综述丹参酮ⅡA对脑缺血后炎性反应影响机制的研究进展,为丹参酮ⅡA的脑缺血保护作用机制研究提供新思路。
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丹参酮ⅡA对脑缺血后炎性反应影响机制的研究进展

doi: 10.3969/j.issn.1006-0111.2018.02.003
    基金项目:  浦东新区卫生系统重要薄弱学科建设资助(PWZbr2017-16)

摘要: 丹参酮ⅡA是从丹参中提取的一种菲醌类衍生物,是丹参发挥脑缺血损伤保护药理作用的主要活性成分之一。炎症在脑缺血发病机制中具有重要作用。近年来许多证据表明,免疫细胞、黏附分子、炎性介质等通过诱导或调控脑缺血时中枢神经系统内免疫应答参与发病过程。综述丹参酮ⅡA对脑缺血后炎性反应影响机制的研究进展,为丹参酮ⅡA的脑缺血保护作用机制研究提供新思路。

English Abstract

冯晶晶, 李铁军, 章越凡. 丹参酮ⅡA对脑缺血后炎性反应影响机制的研究进展[J]. 药学实践与服务, 2018, 36(2): 108-111. doi: 10.3969/j.issn.1006-0111.2018.02.003
引用本文: 冯晶晶, 李铁军, 章越凡. 丹参酮ⅡA对脑缺血后炎性反应影响机制的研究进展[J]. 药学实践与服务, 2018, 36(2): 108-111. doi: 10.3969/j.issn.1006-0111.2018.02.003
FENG Jingjing, LI Tiejun, ZHANG Yuefan. Progress of tanshinone ⅡA on the mechanism of inflammatory response after cerebral ischemia[J]. Journal of Pharmaceutical Practice and Service, 2018, 36(2): 108-111. doi: 10.3969/j.issn.1006-0111.2018.02.003
Citation: FENG Jingjing, LI Tiejun, ZHANG Yuefan. Progress of tanshinone ⅡA on the mechanism of inflammatory response after cerebral ischemia[J]. Journal of Pharmaceutical Practice and Service, 2018, 36(2): 108-111. doi: 10.3969/j.issn.1006-0111.2018.02.003
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