[1] WANG I, WALKER R M, GILLESPIE B M, et al. Risk factors predicting hospital-acquired pressure injury in adult patients: an overview of reviews[J]. Int J Nurs Stud, 2024, 150: 104642. doi:  10.1016/j.ijnurstu.2023.104642
[2] UDESHIKA PRIYADARSHANI SUGATHAPALA R D, LATIMER S, BALASURIYA A, et al. Prevalence and incidence of pressure injuries among older people living in nursing homes: a systematic review and meta-analysis[J]. Int J Nurs Stud, 2023, 148: 104605. doi:  10.1016/j.ijnurstu.2023.104605
[3] CHABOYER W, LATIMER S, PRIYADARSHANI U, et al. The effect of pressure injury prevention care bundles on pressure injuries in hospital patients: a complex intervention systematic review and meta-analysis[J]. Int J Nurs Stud, 2024, 155: 104768. doi:  10.1016/j.ijnurstu.2024.104768
[4] LI Y C, ZHU Z Y, LI S C, et al. Exosomes: compositions, biogenesis, and mechanisms in diabetic wound healing[J]. J Nanobiotechnology, 2024, 22(1): 398. doi:  10.1186/s12951-024-02684-1
[5] AL-MADHAGI H. The landscape of exosomes biogenesis to clinical applications[J]. Int J Nanomedicine, 2024, 19: 3657-3675. doi:  10.2147/IJN.S463296
[6] HE Y Z, YUE J Q, TENG Y W, et al. Tryptanthrin promotes pressure ulcers healing in mice by inhibiting macrophage-mediated inflammation via cGAS/STING pathways[J]. Int Immunopharmacol, 2024, 130: 111687. doi:  10.1016/j.intimp.2024.111687
[7] LUO M, ZHAO F K, CHENG H, et al. Macrophage polarization: an important role in inflammatory diseases[J]. Front Immunol, 2024, 15: 1352946. doi:  10.3389/fimmu.2024.1352946
[8] HUANG X, DENG Y F, XIAO J H, et al. Genetically engineered M2-like macrophage-derived exosomes for P. gingivalis-suppressed cementum regeneration: From mechanism to therapy[J]. Bioact Mater, 2023, 32: 473-487.
[9] QIAN Y F, CHU G L, ZHANG L, et al. M2 macrophage-derived exosomal miR-26b-5p regulates macrophage polarization and chondrocyte hypertrophy by targeting TLR3 and COL10A1 to alleviate osteoarthritis[J]. J Nanobiotechnol, 2024, 22(1): 72. doi:  10.1186/s12951-024-02336-4
[10] CHEN Y L, WANG T, LIANG F X, et al. Nicotinamide phosphoribosyltransferase prompts bleomycin-induced pulmonary fibrosis by driving macrophage M2 polarization in mice[J]. Theranostics, 2024, 14(7): 2794-2815. doi:  10.7150/thno.94482
[11] YANG J J, GONG X Y, LI T, et al. Tantalum particles promote M2 macrophage polarization and regulate local bone metabolism via macrophage-derived exosomes influencing the fates of BMSCs[J]. Adv Healthc Mater, 2024, 13(17): e2303814. doi:  10.1002/adhm.202303814
[12] YUAN Y, TAKASHI E, HOU P, et al. Topical skin application of small-molecule antiplatelet agent against pressure injury in rat models[J]. Int J Mol Sci, 2024, 25(3): 1639. doi:  10.3390/ijms25031639
[13] GHANBARI M, SALKOVSKIY Y, CARLSON M A. The rat as an animal model in chronic wound research: an update[J]. Life Sci, 2024, 351: 122783. doi:  10.1016/j.lfs.2024.122783
[14] YANG J J, WANG X, FAN Y H, et al. Tropoelastin improves adhesion and migration of intra-articular injected infrapatellar fat pad MSCs and reduces osteoarthritis progression[J]. Bioact Mater, 2021, 10: 443-459.
[15] CHEN X T, WAN Z, YANG L, et al. Exosomes derived from reparative M2-like macrophages prevent bone loss in murine periodontitis models via IL-10 mRNA[J]. J Nanobiotechnology, 2022, 20(1): 110. doi:  10.1186/s12951-022-01314-y
[16] CUI Y, HONG S B, XIA Y H, et al. Melatonin engineering M2 macrophage-derived exosomes mediate endoplasmic reticulum stress and immune reprogramming for periodontitis therapy[J]. Adv Sci, 2023, 10(27): e2302029. doi:  10.1002/advs.202302029
[17] SHI L, HU H B, SUN P X, et al. RPL38 knockdown inhibits the inflammation and apoptosis in chondrocytes through regulating METTL3-mediated SOCS2 m6A modification in osteoarth-ritis[J]. Inflamm Res, 2022, 71(7-8): 977-989. doi:  10.1007/s00011-022-01579-x
[18] SMOLOCK E M, KORSHUNOV V A, GLAZKO G, et al. Ribosomal protein L17, RpL17, is an inhibitor of vascular smooth muscle growth and carotid intima formation[J]. Circulation, 2012, 126(20): 2418-2427. doi:  10.1161/CIRCULATIONAHA.112.125971
[19] CHEN G H, REN J G, XIA H F, et al. Ribosomal protein L36-mediated selective loading of microRNA-4432 into extracellular vesicles contributes to perivascular cell dysfunction in venous malformations[J]. Br J Dermatol, 2025, 192(4): 717-727. doi:  10.1093/bjd/ljae492
[20] ZENG J R, ZHANG Y, ZHANG H Y, et al. RPL22 overexpression promotes psoriasis-like lesion by inducing keratinocytes abnormal biological behavior[J]. Front Immunol, 2021, 12: 699900. doi:  10.3389/fimmu.2021.699900